Enzyme Activity and Propitious Fetal Growth


         Dr. Justa Smith together with an ever increasing number of other biochemists and 
microbiologists believe that a number of different enzymes crucial to the development of  
“culture-appropriate” masculine assertiveness, competitiveness and drive, are released 
sometime during the second trimester of pregnancy--if the fetus is male. These 
enzymes permit the testosterones and androgens to work on various sections of the brain 
which nature has programmed to be responsive to these male hormones. In other words, 
assuming healthy enzymes, the male hormones will work to masculinize the brain. If 
one or another enzyme is for any reason malfunctioning, then the part of the brain 
that is specific to that enzyme will not permit the male hormones to do their job.
And that part of the boy fetus’ brain will not be suitably or sufficiently masculinized.
         If the brain of a fetus is left alone, it will develop into a female brain 
regardless of whether or not it is exposed to feminizing hormones. In essence, this is 
one of the myriad reasons why the male is a more delicate organism than the female. 
Many more things can go wrong in male fetal development than in female fetal development. 
Unless the male fetus is exposed to masculinizing hormones plus the enzymes which 
permit each of these hormones to work on various sections of the brain, that male fetus 
will develop a brain that is in at least some ways feminine. The number of ways 
in which it will be feminine will be determined by the number of sections of the developing 
brain that had been deprived of the appropriate masculinizing enzymes. Again, the enzymes
permit the male hormones to do their masculinizing job.
         Now, as I point out in a later chapter, maternal depression, stress, nervousness 
and irascibility of temperament, have been found to impact adversely upon the masculinizing 
enzymes and hormones. This is why tense, nervous, depressed, high strung women are far more 
likely than calm, happy-go-lucky, relaxed women to give birth to children who are passive, 
shy, and noncompetitive. Of course, if such a woman’s fetus is female there is no chance 
that any harm will be done. But if the fetus is male, harm quite typically is done as a 
result of important enzymes being neutralized or enfeebled by the psychosomatic, biochemical
processes caused by the pregnant woman’s irascibility, depression, tense nervousness, etc.
         There are different sections of the fetal brain that need to be masculinized. And 
each of these sections calls for the propitious operation of a different enzyme. Some 
sections of the brain have to do with sexual/romantic directionality. When these brain
sections are inadequately masculinized, the person stands a good chance of becoming a 
pre-homosexual or a pre-bisexual little boy. Other brain sections have to do with 
effeminacy. When these are not masculinized, the person becomes an effeminate little
boy. And contrary to popular impression, most effeminate men are NOT homosexual. 
Because of a specific enzyme deficiency during their prenatal period, they become 
effeminate heterosexuals. And most of them, like heterosexuals generally, will marry and
become fathers. But because of rigid and often uncompromising gender role expectations 
for males, they will suffer much teasing and hazing throughout their formative years as 
a result of their effeminacy. 
         Now, another section of the brain has to do with social assertiveness, competitiveness 
and drive--the opposite of “feminine” passivity. And this is the brain section which has a very 
strong bearing up shyness generally, and especially upon love-shyness
and the behavioral inertia that typically accompanies it. The nonassertive, unaggressive 
little boy will commonly develop non-masculine interest patterns. In essence, he violates 
traditional gender role expectations in terms of interests and preferences rather than in 
terms of either effeminacy or in terms of homosexuality or erotic orientation. For example, 
he will prefer quiet, non-physical forms of play; working with arts and crafts, music and 
theatre arts, dolls and figurines, etc., all of which relate in different ways to violation 
of gender role expectation.
         To be sure, occasionally a number of different enzymes will malfunction while a child is 
intrautero. And in that case the child (if male) will develop a number of different problems. 
For example, he may develop BOTH effeminacy AND homosexuality. Or he may develop BOTH 
effeminacy and chronic shyness. Or he may develop chronic shyness and homosexuality. And in a 
few rare cases he may develop all three of these separate problems. 
         The shy, passive boy is very often mislabeled “homosexual” as the effeminate boy is perhaps 
even more often mislabeled “homosexual”. Conservative and rigid people tend to apply the label 
“homosexual” to any young boy who violates traditional gender role expectations, just as these same 
people commonly affix such labels as  “communist” and “unAmerican” to any person who 
espouses a political, social or religious attitude or belief with which they happen to disagree.
         However, just as the person who believes in the judicious propriety of premarital cohabitation 
is almost never either a “communist” or “antiAmerican” or even “promiscuous”, so the little boy 
who prefers gentle, quiet activities in a coeducational play group is similarly very seldom a 
pre-homosexual. For such a little boy, the enzyme affecting heterosexual directionality worked 
properly. Similarly, the one impacting upon masculinity of demeanor (versus effeminacy) 
probably also worked satisfactorily well. Only the enzyme affecting that part of the brain 
which is associated with masculine assertiveness and competitive drive failed to work well 
enough to permit the appropriate androgens and testosterones to do their brain-masculinizing 
work.